Disorders of Voluntary MuscleCambridge University Press, 21 Ιαν 2010 This major new edition fulfils the need for a single-volume, up-to-date information resource on the etiology, pathogenesis, diagnosis and treatment of diseases of skeletal muscles, including the muscular dystrophies, mitochondrial myopathies, metabolic myopathies, ion channel disorders, and dysimmune myopathies. As background to the clinical coverage, relevant information on advances in molecular and developmental biology, immunopathology, mitochondrial biology, ion-channel dynamics, cell membrane and signal transduction science, and imaging technology is summarized. Combining essential new knowledge with the fundamentals of history-taking and clinical examination, this extensively illustrated book will continue to be the mainstay for practising physicians and biomedical scientists concerned with muscle disease. Regular updates on the clinical and basic science aspects of muscle disease - written mainly by rising stars of myology - will be published on an accompanying website. |
Αναζήτηση στο βιβλίο
Αποτελέσματα 1 - 5 από τα 67.
Σελίδα
... identified histochemically. The glycogen depletion method showed that muscle fibers belonging to the same motor unit were arranged in a mosaic fashion among muscle fibers belonging to othermotorunits [54, 55].Relatively fewmuscle ...
... identified histochemically. The glycogen depletion method showed that muscle fibers belonging to the same motor unit were arranged in a mosaic fashion among muscle fibers belonging to othermotorunits [54, 55].Relatively fewmuscle ...
Σελίδα
... . The satellite cell is a resident quiescent mononucleated myogenic precursor cell located on the surface of the myofiber beneath the basal lamina. Figure 2.2. Satellite cells identified by electron microscopy. High magnification.
... . The satellite cell is a resident quiescent mononucleated myogenic precursor cell located on the surface of the myofiber beneath the basal lamina. Figure 2.2. Satellite cells identified by electron microscopy. High magnification.
Σελίδα
... identified by electron microscopy. High magnification of satellite cells/myoblasts shown by transmission electron microscopy, in regenerating adult mouse muscle sampled up to 5 days after chemical injury. (a) Classical quiescent ...
... identified by electron microscopy. High magnification of satellite cells/myoblasts shown by transmission electron microscopy, in regenerating adult mouse muscle sampled up to 5 days after chemical injury. (a) Classical quiescent ...
Σελίδα
... identified until a basal lamina can be detected and this is around 10– 15 weeks in utero in humans [8]. Genes and signaling pathways involved in the transition from a population of fetal muscle precursor cells to a selfrenewing ...
... identified until a basal lamina can be detected and this is around 10– 15 weeks in utero in humans [8]. Genes and signaling pathways involved in the transition from a population of fetal muscle precursor cells to a selfrenewing ...
Σελίδα
... identified bytheiranatomical positionusing electron microscopy (Figure 2.2). Even thiscan bedifficult sincepericytes can resemble satellite cells,and macrophages,neutrophils and othercellscan infiltrate and lie beneaththe basallaminaof ...
... identified bytheiranatomical positionusing electron microscopy (Figure 2.2). Even thiscan bedifficult sincepericytes can resemble satellite cells,and macrophages,neutrophils and othercellscan infiltrate and lie beneaththe basallaminaof ...
Περιεχόμενα
Dedication Section scientific basis | |
Dystrophic myopathies of early childhood onset | |
Investigation of muscle disease 4 Electrophysiological evaluation of suspected myopathy | |
Eugenio MercuriandMarianne deVisser Section 3A Descriptionofmuscledisease general aspects 8 The clinical assessmentanda guide to classification o... | |
The congenitalmyopathies Carina WallgrenPettersson and NigelG Laing 14 Muscle diseases with prominent muscle contractures | |
Defects ofcarbohydrate andlipid | |
Muscular dystrophies presenting with proximal muscle | |
Muscle ionchannelopathies and related disorders | |
Συχνά εμφανιζόμενοι όροι και φράσεις
abnormalities actin activity andthe antibodies associated associatedwith atrophy autosomal dominant Biol Brain canbe cardiac cardiomyopathy caused caveolin3 chromosome clinical CMAP collagen complex congenital muscular dystrophy congenital myopathies contractures defects deficiency dehydrogenase deletions denervation dermatomyositis diagnosis disorders distal myopathy Duchenne muscular dystrophy dysferlin dystroglycan encoding enzyme etal exon expression facioscapulohumeral facioscapulohumeral muscular dystrophy Figure filaments FSHD function gene Genet glycogen glycosylation identified inclusion body myositis inflammatory myopathies involvement isoforms Karpati lamina LGMD LGMD2I limbgirdle muscular dystrophy membrane metabolic mice missense mitochondrial molecular motor unit mtDNA muscle biopsy muscle disease muscle fibers Muscle Nerve muscle weakness musclefibers mutationsin myoblasts myofiber myofibrillar myosin myotilin myotonia myotonic dystrophy nemaline myopathy Neurol Neurology neuromuscular normal nuclear nuclei ofmuscle ofthe onset pathology patients patientswith phenotype protein receptor recessive regeneration respiratory RYR1 sarcoglycan sarcolemma sarcomere satellite cells skeletal muscle staining syndrome therapy tissue utrophin vacuoles Xlinked